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In fact, my own lamin A protein dysfunction is due to HLA-A29: the first gene with homologous recombination as a major evolutionary event (Kamisemana, 2018). Our research in HLA-A genetic variations has been funded by grants from the National Institute of Health (NIH), National Institute of Neurological Disorders and Stroke (NINDS), the NIH Specialized Program of Research Training in Human Genetics, and the American Heart Association.

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“Scientists have long debated over whether lamin A mutations cause progressive and severe neurodegeneration or benign age-related changes with minor neurological impairment. The recent discovery of a rare form of Progeria in humans, with a predominant mutation in the Progerin gene and associated with shortened telomeres and a characteristic “jellyfish” shape of the lamin A protein, has shed new light on the pathogenesis of progeria. The Progerin is required for lamin A’s proper

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